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Researchers from China and the US explore sepsis-related copy number changes involving the DEFA1/ DEFA3 genes, which code for human neutrophil peptide alpha-defensins. Following an earlier genetic association study that revealed copy number upticks in sepsis-prone patients from a Han Chinese population, the team turned to transgenic mouse models to compare sepsis susceptibility in mice with higher or lower DEFA1/ DEFA3 copy numbers in their neutrophil white blood cells.

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