New Research Points to miR-25 as a Therapeutic Target for Heart Failure | GenomeWeb

A group led by researchers from Sanford-Burnham Medical Research Institute published data last week showing that inhibiting a single microRNA, miR-25, could stop the progression of heart failure in a mouse model while boosting the animals' cardiac function and survival.

The findings implicate miR-25 as a key player in the decline of cardiac function during heart failure and demonstrate its promise as a therapeutic target for the condition.

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