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IP Update: Recent Patents Awarded to Novozymes, University of Texas, and More

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Title: Methods for Eliminating or Reducing the Expression of a Gene in a Filamentous Fungal Strain

Patent Number: 8,716,023

Filed: Dec. 9, 2004

Lead Inventor: Howard Brody, Novozymes


Title: microRNA Compositions and Methods for the Treatment of Myelogenous Leukemia

Patent Number: 8,716,255

Filed: Aug. 11, 2008

Lead Inventor: Florian Kuchenbauer, British Columbia Cancer Agency


Title: Compositions and Methods for Silencing Ebola Virus Gene Expression

Patent Number: 8,716,464

Filed: July 20, 2010

Lead Inventor: Thomas Geisbert, University of Texas Medical Branch (Tekmira Pharmaceuticals)


Title: Kit for Making Induced Pluripotent Stem Cells using Modified RNAs

Patent Number: 8,716,465

Filed: Aug. 21, 2012

Lead Inventor: Derrick Rossi, Children's Medical Center


Title: microRNAs for Identification of Exposure to Lower Aliphatic Saturated Aldehydes and the Method of Identification using Thereof

Patent Number: 8,716,192

Filed: Nov. 29, 2012

Lead Inventor: Jae Chun Ryu, Korea Institute of Science and Technology


Title: Regulation of Metabolism by miR-378

Patent Number: 8,716,258

Filed: June 6, 2011

Lead Inventor: Eric Olson, University of Texas

The Scan

Germline-Targeting HIV Vaccine Shows Promise in Phase I Trial

A National Institutes of Health-led team reports in Science that a broadly neutralizing antibody HIV vaccine induced bnAb precursors in 97 percent of those given the vaccine.

Study Uncovers Genetic Mutation in Childhood Glaucoma

A study in the Journal of Clinical Investigation ties a heterozygous missense variant in thrombospondin 1 to childhood glaucoma.

Gene Co-Expression Database for Humans, Model Organisms Gets Update

GeneFriends has been updated to include gene and transcript co-expression networks based on RNA-seq data from 46,475 human and 34,322 mouse samples, a new paper in Nucleic Acids Research says.

New Study Investigates Genomics of Fanconi Anemia Repair Pathway in Cancer

A Rockefeller University team reports in Nature that FA repair deficiency leads to structural variants that can contribute to genomic instability.