The P. Brundin lab focus on pathogenetic mechanisms and pharmacological treatment in cell and animal models of Parkinson's disease. They also study cell replacement therapy with stem cells in attempts to repair brains in animal models of parkinsonism. The group's mission is to understand neurodegenerative diseases and develop new therapies that are of benefit to patients and their caregivers.
Specifically, we are interested in candidates with a background in neuroscience and Parkinson’s disease.
The postdoctoral position is available in the winter/spring of 2017 and is for a motivated scientist who meets the qualifications and description outlined below. The fellow will investigate the contribution of alpha-synuclein to the pathophysiology of Parkinson’s disease in pre-clinical model systems.
The overall goal of the project is to study the contribution of alpha-synuclein to Parkinson’s disease.The candidate will use established model systems to determine effects of alpha-synuclein pathology. The results from these studies could lead to development of novel therapeutic strategies to improve clinical outcomes of Parkinson’s disease.