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Adaptive Complexity talks about a paper published this week in PNAS that lends a clue to why cancer cells can keep growing and dividing, even in an environment that lacks nutrients. Princeton's David Botstein used a model of "unnaturally" starved mutant yeast to find mutations in the TOR pathway, which is a critical stress response regulatory pathway.

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Bioethicists disagree with a research team's decision to allow the return of risk results for adult-onset conditions from a newborn sequencing project, according to Reuters.

Alterations to particular gene may enable the Quechua of Peru to better tolerate high-altitude life, Ars Technica reports.

Nature News reports that additional South Korean researchers have included the names of children on scientific papers when they did not contribute to the work.

In PLOS this week: statistical approach to prioritize rare variant searches, gene expression alterations in chronic obstructive pulmonary disease, and more.