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Yeast and Cancer: Similar But Different?

Adaptive Complexity talks about a paper published this week in PNAS that lends a clue to why cancer cells can keep growing and dividing, even in an environment that lacks nutrients. Princeton's David Botstein used a model of "unnaturally" starved mutant yeast to find mutations in the TOR pathway, which is a critical stress response regulatory pathway. Follow-up studies would compare these mutations to potentially similar ones in cancer cells, which, like yeast that have been genetically modified to not be able to produce certain nutrients for themselves, will nonetheless keep growing and dividing.

The Scan

Gone, But Now Reconstructed SARS-CoV-2 Genomes

In a preprint, a researcher describes his recovery of viral sequences that had been removed from a common database.

Rare Heart Inflammation Warning

The Food and Drug Administration is adding a warning about links between a rare inflammatory heart condition and two SARS-CoV-2 vaccines, Reuters reports.

Sandwich Sampling

The New York Times sent tuna sandwiches for PCR analysis.

Nature Papers Describe Gut Viruses, New Format for Storing Quantitative Genomic Data, More

In Nature this week: catalog of DNA viruses of the human gut microbiome, new dense depth data dump format to store quantitative genomic data, and more.