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In an advance, online publication of Nucleic Acids Research this week, an international research team describes a novel role for miR-382, which they've uncovered using miRNA-target pairs in human renal epithelial cells treated with transforming growth factor β1. "Knockdown of miR-382, which was upregulated by TGFβ1, attenuated TGFβ1-induced loss of the epithelial marker E-cadherin," while "knockdown of miR-382 attenuated TGFβ1-induced downregulation of SOD2," the authors write.

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