Technology Review reports on research that may yield insight into aging. "A new study by Harvard Medical School researchers reveals that the biochemical mechanism that makes yeast grow old has a surprising parallel in mice, suggesting it may be a universal cause of aging in all organisms," the article says. In yeast, the Sir2 protein has long been implicated in aging; this new study shows that the mouse equivalent, SIRT1, has a similar role. David Sinclair was senior author of the paper, which just came out in Cell. He's quoted in the story as saying, "It was very exciting when we made the discovery, because it was so unexpected."
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