Skip to main content
Premium Trial:

Request an Annual Quote

Invitrogen to Make Sequence Analysis Software Free for Nonprofit Labs

NEW YORK, Sept. 28 (GenomeWeb News) - Invitrogen has created a Vector NTI Open Access policy and the online Vector NTI User Community in conjunction with the release of Vector NTI Advance 10, the latest version of its NTI sequence analysis software, the firm said today.


According to the company, the Open Access policy will enable researchers in not-for-profit laboratories to apply for free, annual, and renewable licenses of Vector NTI. The User Community is designed to serve as an online meeting place for researchers to acquire the latest versions of the software, obtain technical resources, and communicate with Invitrogen and other users, Invitrogen said.


Greg Lucier, chairman and CEO of Invitrogen, said the company anticipates "having tens of thousands of users in the academic, government and research institutions take advantage of our Open Access policy."

Invitrogen said that the Wellcome Trust Sanger Institute and The Scripps Research Institute are both Open Access participants.


GenomeWeb News' sister publication BioInform reported on the developments surrounding Vector NTI two weeks ago.

The Scan

Germline-Targeting HIV Vaccine Shows Promise in Phase I Trial

A National Institutes of Health-led team reports in Science that a broadly neutralizing antibody HIV vaccine induced bnAb precursors in 97 percent of those given the vaccine.

Study Uncovers Genetic Mutation in Childhood Glaucoma

A study in the Journal of Clinical Investigation ties a heterozygous missense variant in thrombospondin 1 to childhood glaucoma.

Gene Co-Expression Database for Humans, Model Organisms Gets Update

GeneFriends has been updated to include gene and transcript co-expression networks based on RNA-seq data from 46,475 human and 34,322 mouse samples, a new paper in Nucleic Acids Research says.

New Study Investigates Genomics of Fanconi Anemia Repair Pathway in Cancer

A Rockefeller University team reports in Nature that FA repair deficiency leads to structural variants that can contribute to genomic instability.